[1] |
王志,张成,王俭,等. GerdQ量表在胃食管反流病诊断中的应用[J/CD].中华胃食管反流病电子杂志, 2014, 1(1):36-38.
|
[2] |
杜智,张成,克力木·阿不都热依木,等.高分辨率食管测压与食管24h pH监测在胃食管反流病中的应用价值[J/CD].中华胃食管反流病电子杂志, 2015, 2(3):147-151.
|
[3] |
瞿国强.胃食管反流病的流行病学研究现状[J].中国临床保健杂志, 2011, 14(6):660-663.
|
[4] |
韩梦婷,李治建,等.胃食管反流病的研究进展[J].中国当代医药, 2017, 24(25):13-16.
|
[5] |
Saegusa. Y, Takeda H, Muto S, et al, Decreased motility of the lower esophageal sphincter in a rat model of gastroesophageal reflux disease may be mediated by reductions of serotonin and acetylcholine signaling [J]. Biol Pharm Bull, 2011, 34(5):704-711.
|
[6] |
Farré R, De Vos R, Geboes K, et al, Critical role of stress in increased oesophageal mucosa permeability and dilated intercellular spaces[J]. Gut, 2007, 56(9):1191.
|
[7] |
Knowles CH, Aziz O. Visceral hypersensitivity in non-erosive reflux disease [J]. Gut, 2008, 57(5):674.
|
[8] |
谭佳成,叶必星,林琳,等,胃食管反流病相关的细胞间隙增宽和紧密连接蛋白[J].胃肠病学, 2013. 18(10):636-638.
|
[9] |
Weijenborg PW, Smout AJ, Verseijden C, et al. Hypersensitivity to acid is associated with impaired esophageal mucosal integrity in patients with gastroesophageal reflux disease with and without esophagitis [J]. Am J Physiol Gastrointest Liver Physiol, 2014, 307(3):G323-329.
|
[10] |
刘荣泉,那吉,陶家丽,等.贲门松弛与胃食管反流病患者的食管下段DIS改变[J].胃肠病学和肝病学杂志, 2016, 25(1):62-65.
|
[11] |
Woodland P, Al-Zinaty M, Yazaki E, et al , In vivo evaluation of acid-induced changes in oesophageal mucosa integrity and sensitivity in non-erosive reflux disease [J]. Gut, 2013,62(9):1256.
|
[12] |
Souza RF, Huo X, Mittal V, et al , Gastroesophageal reflux might cause esophagitis through a cytokine-mediated mechanism rather than caustic acid injury [J]. Gastroenterology, 2009, 137(5):1776-1784.
|
[13] |
Calabrese C, Fabbri A, Bortolotti M, et al , Dilated intercellular spaces as a marker of oesophageal damage: comparative results ingastro-oesophageal reflux disease with or without bile reflux [J]. Aliment Pharmacol Ther,2003,18(5):525-532.
|
[14] |
徐姝燕,富建华.紧密连接相关蛋白Occludin的研究进展[J].国际儿科学杂志, 2012, 39(5):451-454.
|
[15] |
陈意,李方方,张勇,等.肠黏膜上皮组织紧密连接的生物学功能和作用机理[J].动物营养学报, 2017, 29(9):3068-3074.
|
[16] |
Van Itallie CM, Anderson JM. Architecture of tight junctions and principles of molecular composition [J]. Semin Cell Dev Biol, 2014, 36:157-165.
|
[17] |
Jovov B, Que J, Tobey NA, et al. Role of E-cadherin in the pathogenesis of gastroesophageal reflux disease[J]. Am J Gastroenterol, 2011, 106(6):1039-1047.
|
[18] |
雷军,夏祥国.紧密连接蛋白claudin的研究进展[J].西南医科大学学报, 2009, 32(6):655-657.
|
[19] |
闫洪领,王季猛.胞质紧密粘连蛋白1的研究现状[J].医学综述, 2008, 14(17):2589-2591.
|
[20] |
刘贝妮,姚树坤.食管黏膜屏障功能及其检测方法研究进展[J].胃肠病学和肝病学杂志, 2014, 23(12):1373-1375.
|
[21] |
周童,仵敏娟.连接黏附分子A的研究进展[J].生命科学, 2013(9):865-870.
|
[22] |
Yoshida N. Inflammation and oxidative stress in gastroesophageal reflux disease[J]. J Clin Biochem Nutr, 2007,40:13-23.
|
[23] |
Mönkemüller K, Wex T, Kuester D, et al. Interleukin-1Beta and Interleukin-8 Expression Correlate with the Histomorphological Changes in Esophageal Mucosa of Patients with Erosive and Non- Erosive Reflux Disease [J]. Digestion, 2009. 79(3):186-195.
|
[24] |
Oh TY, Lee JS, Ahn BO, et al. Oxidative damages are critical in pathogenesis of reflux esophagitis: implication of antioxidants in its treatment [J]. Free Radical Bio Med, 2001, 30(8):905-915.
|
[25] |
Chen H, Hu Y, Fang Y, et al. Nrf2 deficiency impairs the barrier function of mouse esophageal epithelium [J]. Gut, 2014,63(5):711-719.
|
[26] |
Wakabayashi N, Itoh K, Wakabayashi J, et al, Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation[J]. Nature Genetics,2003, 35(3):238-245.
|
[27] |
Fang Y, Chen H, Hu Y, et al. Gastroesophageal reflux activates the NF-κB pathway and impairs esophageal barrier function in mic[J]. Am J Physiol-Gastr L, 2013. 305(1):pG58.
|
[28] |
Li FY, Li Y. Interleukin-6, desmosome and tight junction protein expression levels in reflux esophagitis-affected mucosa[J]. World J Gastroenterol, 2009, 15(29):3621-3630.
|